There is more than one way to present the variety of congenital heart diseases. short segment severe aortic coarctation.General differential considerations include: Surgical correction (either single- or multistage) is the definitive treatment. Prostaglandin E 1 may be given to initial management to keep the ductus open. If uncorrected, it carries a very poor prognosis with extrauterine survival being as little as a few days. great vessels may show a "V" configuration on coronal imaging 2.non-visualisation of the portion of interruption.CTĪllows visualisation of the interrupted aortic arch and associated anomalies. These modalities may not allow differentiation of IAA from severe aortic coarctation with a hypoplastic arch 9. The ascending aorta may also appear more vertical than usual. The right ventricle may appear a lot larger than the left, although this is a non-specific finding. Plain film features are often non-specific 3: aortopulmonary septal defect (aortopulmonary window).almost always associated if there is a right-sided descending aorta.found commonly in those with a type B interruption.subtype 3: isolated subclavian artery that arises from the ductus arteriosus.type C: rare, interruption occurs proximal to the left common carotid arterial originĮach type is divided into three subtypes 7:.type B: most common (>50%), the break occurs between the left common carotid and left subclavian arterial origins.type A: second most common, the interruption occurs distal to the left subclavian arterial origin.ClassificationĪccording to the Celoria-Patton classification, IAA can be classified into three types according to the location of the anomaly: Pathologyįaulty embryological development of the aortic arch (thought to occur during the 5 th to 7 th week of intrauterine life). It may account for ~1.5% of congenital cardiac anomalies.
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